Covid vaccines encode not only viral spike protein but also two mutated genes to virilize girls, cause ovarian failure and cancer: purported Moderna engineers
Adjuvants secretly added to the vaccine enhance reverse transcription of vaccinal mRNA to heritable DNA, claim ostensible whistleblowers
Two alleged Moderna engineers (not pictured above) exposed potentially alarming information on 4chan in December 2020 — when Covid vaccines were just starting to roll out — about two mutated genes they claim are secretly encoded by vaccinal mRNA.
The purported Moderna whistleblowers say these mutated genes have been placed in vaccines to cause 1) premature ovarian failure in girls, sterilizing them; and 2) cancer in both men and women decades after injection.
If true, one of these mutated autosomal recessive genes could cause one-fourth of daughters born of vaccinated parents to have ambiguous genitalia, virilization (beard growth (hirsutism) and deepened voice), acne, polycystic ovary syndrome, and/or premature ovarian failure.
This inheritance frequency of one-fourth assumes each parent has only one copy of an affected gene in their sperm or eggs. If one parent has two copies and the other parent has one, half their daughters would be affected. If both parents have two copies, all their daughters would be.
Boys can also be affected by this gene mutation due to a resultant lack of estrogen, involved in bone development and glucose metabolism, among other things.
Possibly credible allegation
The supposed whistleblowers’ information is perhaps credible because it describes specific genetic principles an average prankster would be unlikely to know.
On the other hand, this could be intelligence agency disinformation to label people who inquire about it as “antivaxxers” and “conspiracy theorists.”
To confirm these allegations or rule them out as false, a quick investigation is warranted, at least by testing somatic (body) cells in both vaccinated men and women, e.g., by a cheek swab, or, if necessary, by testing sperm of vaccinated men.
There is no need to harvest ovarian follicles for this testing, based on what we know so far.
Allegedly mutated genes
Here are the two genes mentioned:
CYP19A1 (aromatase gene, converting testosterone to estradiol)
The CYP19A1 gene provides instructions for making an enzyme called aromatase. This enzyme converts a class of hormones called androgens like testosterone, involved in male sexual development, to different forms of the female sex hormone estrogen.
The activity (expression) of aromatase varies among different cell types depending on the cells' need for estrogen. In females, aromatase is most active in the ovaries, where it guides sexual development. In males, aromatase is most active in fat (adipose) tissue. In both males and females, estrogen plays a role in regulating bone growth and blood sugar levels. During fetal development, aromatase converts androgens to estrogens in the placenta, which is the link between the mother's blood supply and the fetus. This conversion in the placenta prevents androgens from directing sexual development in female fetuses. After birth, the conversion of androgens to estrogens takes place in multiple tissues.
CDKN1B (Cyclin Dependent Kinase Inhibitor 1B, a tumor suppressor gene)
Important regulator of cell cycle progression, on Chromosome 12. Inhibits the kinase activity of CDK2 bound to cyclin A. Involved in G1 [cell cycle] arrest. Overexpressed with CCND3 in agressive B cell lymphomas, underexpressed in the development and progression of oral squamous cell carcinoma and in thyroid, colon, breast, prostate, and superficial bladder carcinomas, deleted in leukemias and other cancers with poor prognosis (pancreatic adenocarcinomas).
The CDKN1B gene provides instructions for making a protein called p27. This protein is found in cells and tissues throughout the body. Within cells, p27 is located primarily in the nucleus, where it plays a critical role in controlling cell growth and division. It helps regulate the cell cycle, which is the cell's way of replicating itself in an organized, step-by-step fashion. Specifically, p27 normally blocks cells from entering the phase of the cell cycle when DNA is copied (replicated) in preparation for cell division. By blocking cell cycle progression, p27 prevents cells from dividing too quickly or at the wrong time. Based on this function, p27 is described as a tumor suppressor protein. Studies suggest that p27 is also involved in controlling cell differentiation, which is the process by which cells mature to carry out specific functions.
Other diseases from these genes
Many other problems can occur from mutations in these genes besides ovarian failure, assuming a child is homozygous; i.e., they have two gene copies, one each from their vaccinated mother and father.
List of diseases associated with this gene
Diseases related to aromatase deficiency:
osteopenia and osteoporosis (reduced bone mass)
delayed skeletal maturation
genu valgum (“knock knees”)
cryptorchidism (undescended testes)
amenorrhea (loss of menstrual bleeding)
CDKN1B (Cyclin Dependent Kinase Inhibitor):
List of diseases associated with this gene